Beta hexosaminidase a hi-res stock photography and images

Tay-sachs Disease, illustration Stock Photo

RM2BEH9B7–Tay-sachs Disease, illustration

Molecule of beta-hexosaminidase A enzyme, or HEXA, illustration. Mutations in the gene encoding HEXA decrease the hydrolysis of GM2 gangliosides, which is the main cause of Tay-Sachs disease. Stock Photo

RF2HBA1BN–Molecule of beta-hexosaminidase A enzyme, or HEXA, illustration. Mutations in the gene encoding HEXA decrease the hydrolysis of GM2 gangliosides, which is the main cause of Tay-Sachs disease.

Crystallographic structure of human beta-Hexosaminidase A. 3D cartoon and Gaussian surface models, PDB 2gjx, white background Stock Photo

RF2M98556–Crystallographic structure of human beta-Hexosaminidase A. 3D cartoon and Gaussian surface models, PDB 2gjx, white background

RM2BEH9CH–Tay-sachs Disease, illustration

RF2HBA1BD–Molecule of beta-hexosaminidase A enzyme, or HEXA, illustration. Mutations in the gene encoding HEXA decrease the hydrolysis of GM2 gangliosides, which is the main cause of Tay-Sachs disease.

Tay-Sachs disease, computer illustration Stock Photo

RF2HGM8HM–Tay-Sachs disease, computer illustration

RF2HGM8H7–Tay-Sachs disease, computer illustration

RF2HGM8HD–Tay-Sachs disease, computer illustration

RF2HGM8HC–Tay-Sachs disease, computer illustration

RF2HGM8HJ–Tay-Sachs disease, computer illustration

RF2HGM8GY–Tay-Sachs disease, computer illustration

RF2HGM8HT–Tay-Sachs disease, computer illustration

RF2HGM8HB–Tay-Sachs disease, computer illustration

Tay-Sachs disease, illustration. A genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability. Stock Photo

RF2HBA1FH–Tay-Sachs disease, illustration. A genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1DP–Tay-Sachs disease, illustration. A genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1D6–Tay-Sachs disease, illustration. A genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability. Stock Photo

RF2HBA1EF–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1DY–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1E7–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes, myelin degradation. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1E6–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes, myelin degradation. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1DF–Tay-Sachs disease, illustration. A genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1DN–Tay-Sachs disease, illustration. A genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1DK–Tay-Sachs disease, illustration. A genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1DG–Tay-Sachs disease, illustration. A genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

Illustration of Tay-Sachs disease, a genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Neurons become swollen with lamellar inclusions due to accumulation of gangliosides in lysosomes with subsequent neuronal degeneration. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability. Stock Photo

RF2HBA1ER–Illustration of Tay-Sachs disease, a genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Neurons become swollen with lamellar inclusions due to accumulation of gangliosides in lysosomes with subsequent neuronal degeneration. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1E5–Illustration of Tay-Sachs disease, a genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Neurons become swollen with lamellar inclusions due to accumulation of gangliosides in lysosomes with subsequent neuronal degeneration. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1DT–Tay-Sachs disease, illustration. A genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Neurons become swollen with lamellar inclusions due to accumulation of gangliosides in lysosomes with subsequent neuronal degeneration. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1EN–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1EK–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1E9–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1E0–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1EW–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1EC–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes, myelin degradation. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1E8–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes, myelin degradation. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1E4–Neurons in Tay-Sachs disease. Illustration showing swollen neurons with membraneous lamellar inclusions due to accumulation of gangliosides in lysosomes, myelin degradation. Tay-Sachs disease is a disorder that progressively destroys brain neurons, is caused by a mutation in the HEXA gene of chromosome 15 leading to deficiency of hexosaminidase A. Tay-sachs is most commonly seen in infants, manifesting in muscle weakness and decreased motor function, vision and hearing loss, and intellectual disability.

RF2HBA1EH–Illustration of Tay-Sachs disease, a genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromo

RF2HBA1EE–Illustration of Tay-Sachs disease, a genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromo

RF2HBA1EM–Illustration of Tay-Sachs disease, a genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromo

RF2HBA1EY–Illustration of Tay-Sachs disease, a genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromo

RF2HBA1EJ–Illustration of Tay-Sachs disease, a genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromo

RF2HBA1EB–Illustration of Tay-Sachs disease, a genetic disorder that progressively destroys brain neurons. It is caused by a mutation in the HEXA gene of chromo

Beta hexosaminidase a Stock Photos and Images

Beta hexosaminidase a Stock Photos and Images

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