Fibroids and allied tumours (myoma and adenomyoma) : their pathology, clinical features and surgical treatment . Figure 92. Same section as seen above, showing small endotheliallymph-spaces in hyaline areas (X 4). F.ACE I-AGE 79 INFECTION 79 to show as four) show dark areas of telangiectatic change. Figu re 91, Plate XXIV., shows a telangiectatic condition associated with a lymphangiectatic development in a myoma. In Figure 92, Plate XXIV., is seen the microscopic slide (magnified four times) from which Figure 91 was drawn ; it. Fig. 9c.—Showing telangiectatic areas in myomas. The colour has b

Fibroids and allied tumours (myoma and adenomyoma) : their pathology, clinical features and surgical treatment . Figure 92. Same section as seen above, showing small endotheliallymph-spaces in hyaline areas (X 4). F.ACE I-AGE 79 INFECTION 79 to show as four) show dark areas of telangiectatic change. Figu re 91, Plate XXIV., shows a telangiectatic condition associated with a lymphangiectatic development in a myoma. In Figure 92, Plate XXIV., is seen the microscopic slide (magnified four times) from which Figure 91 was drawn ; it. Fig. 9c.—Showing telangiectatic areas in myomas. The colour has b Stock Photo
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Fibroids and allied tumours (myoma and adenomyoma) : their pathology, clinical features and surgical treatment . Figure 92. Same section as seen above, showing small endotheliallymph-spaces in hyaline areas (X 4). F.ACE I-AGE 79 INFECTION 79 to show as four) show dark areas of telangiectatic change. Figu re 91, Plate XXIV., shows a telangiectatic condition associated with a lymphangiectatic development in a myoma. In Figure 92, Plate XXIV., is seen the microscopic slide (magnified four times) from which Figure 91 was drawn ; it. Fig. 9c.—Showing telangiectatic areas in myomas. The colour has been preserved byfixation by Kaiserling-Picks method of preservation. shows the small lymph-channels, in pale connective-tissueareas of the growth, where hyaline change was marked. 9. Infection of Myoma.^—Myomas are liable to infec-tion from three sources. The usual channel by which infec-tion gains access is the vagina and uterine cavity ; thereforethis complication is most commonly met with in submucousgrowths. A second source is also the result of an ascending J.. 8o SECONDARY CHANGES IN MYOMA chap. infection which has now reached the abdominal ostium ofthe tubes, and the myoma thus becomes infected from belowand from above. The third source is the bowel, throughthe adherent peritoneal coat. Myomas may also be in-fected through the blood, and Cullen ^ mentions the caseof an interstitial myoma, globular in shape and 7 cm. indiameter, which was very soft and putty-like on section, smear-preparations from which showed